Re-programming RNA translation

Selective translation control via RNA-guided proximity.

Weld Pharmaceuticals is building a small-molecule platform to tune protein production with selectivity that emerges from biology, enabling targeted effects while aiming to preserve global translation.

RNA-guided induced proximity
Transcript-selective effects
Small-molecule drug properties
Modular chemistry

Technology

RIVET platform

RIVET is a small-molecule modality that controls translation through RNA-guided induced proximity, designed to enable selective suppression of marked transcripts while leaving global translation largely intact.

Local, not global

Translation control should be local

Biology already differentiates transcripts through RNA-binding proteins and RNA structure, creating “addresses” that can be leveraged to focus effects where they matter.

Proximity logic

RNA-guided induced proximity

By bringing functional elements together at specific RNA–protein assemblies, we aim to achieve tunable, reversible modulation without requiring destructive mechanisms.

Small molecules

Drug-like delivery

We prioritize small-molecule properties suitable for scalable manufacturing and clinically practical dosing, while building flexibility for follow-on programs.

Approach

Built on converging capabilities

Our approach integrates a chemistry toolkit, transcriptome-scale RNA maps, and validated induced-proximity paradigms to make translation control tractable in drug discovery.

Chemistry toolkit

We focus on ligandable components and modular linker strategies to explore design space efficiently, while protecting proprietary details off-site.

RNA maps

Transcriptome-scale datasets enable a hypothesis-driven view of RNA–protein binding “ZIP codes,” supporting selectivity considerations early in discovery.

Proximity paradigm shift

Induced proximity has been clinically de-risked across modalities, and non-degrading proximity is emerging as a powerful way to modulate function with precision.

Differentiation

Why RIVET

Selectivity emerges from biology, not synthetic design alone, and the modality is designed to be reversible, tunable, and non-destructive.

Versus broad translation inhibitors

Traditional approaches can be limited by global translation shutdown and toxicity. Our objective is localized pharmacology at specific RNA–protein assemblies.

Versus oligonucleotide approaches

We aim for small-molecule-like PK/PD and manufacturability while still addressing transcript-level control, without requiring sequence-dependent therapeutic formats.

Versus degraders

For short-lived proteins, resynthesis can outpace degradation. Translation control can directly modulate production rates and enable rapid functional effects.

Contact

Pre-seed

What we share publicly

We keep this site intentionally high-level while we build. Detailed scientific materials are available under NDA.

Email

info@weldpx.com

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